John H. Morrison, Ph.D.

Neuroscience and Behavior Unit
CNPRC Director & Core Scientist

Professor
Department of Neurology
UC Davis School of Medicine

Research Interest

Dr. Morrison’s research program focuses primarily on the neurobiology of aging and neurodegenerative disorders, particularly as they relate to cellular and synaptic organization of the cerebral cortex. His lab is developing a comprehensive model of synaptic health in the cerebral cortex and how molecular and structural deviations from this profile induced by age and endocrine disruption impact cognitive performance. Dr. Morrison is also investigating the degree to which age-related alterations in structural and molecular attributes of the synapse that lead to cognitive decline leave the brain vulnerable to Alzheimer’s Disease.

Education

Johns Hopkins University School of Medicine
Department of Cell Biology and Anatomy
Ph.D., Neurobiology, 1979

Johns Hopkins University
Baltimore, Maryland
B.A., 1974

Post-Doctoral Training

The Salk Institute
Laboratory of Dr. Floyd E. Bloom
Postdoctoral Fellow, 1980-1982

Johns Hopkins University School of Medicine
Laboratory of Dr. Mark E. Molliver
Departments of Neuroscience and Cell Biology and Anatomy
Postdoctoral Fellow, 1979-1980

Professional Memberships

  • National Academy of Medicine (elected 2016)
  • Society for Neuroscience (elected member of Council, 2010-2013)
  • The Harvey Society (President-Elect, 2009-2010; President, 2010-2011)
  • American Association for the Advancement of Science
  • Association of Medical School Neuroscience Department Chairpersons (AMSNDC), 2002-2006
  • Cajal Club: President – Elect 2000-2002; President 2002-2004; Past President, 2004-2006.
  • New York Academy of Science, Section of Neuroscience, Co-Chair 2000-2002
  • New York City Chapter of Society for Neuroscience, Co-Chair, 2000-2002

Email Dr. Morrison

Visit Dr. Morrison’s UC Davis webpage

Media

Neurobiology of Aging

Dr. Morrison's overall goal is to develop interventions that promote cognitive health and prevent Alzheimer’s disease through sustaining synaptic health.